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Yusuke Takezawa

Section

Graduate School of Medicine

Job title

Senior Assistant Professor

Degree

PhD (Tohoku University Graduate School of Medicine)

researchmap

https://researchmap.jp/yusuke.takezawa1983

J-GLOBAL ID

201801015058944314

e-Rad No.

10837793

ORCID

https://orcid.org/0000-0002-2185-860X

Research History 9

2023/07 - 2025/09

Tohoku University　University Hospital Pediatrics
2020/07 - 2023/05

The University of Iowa　Carver College of Medicine, Stead Family Department of Pediatrics　Research Scholar
2020/05 - 2020/06

東北大学病院　小児病態学分野　助教
2019/04 - 2020/04

東北大学病院　小児病態学分野　医員
2018/10 - 2019/03

宮城県立こども病院　神経科　医長
2010/04 - 2013/03

独立法人国立成育医療研究センター病院　総合診療部　レジデント
2008/04 - 2010/03

Ishinomaki Red Cross Hospital　Resident
2025/10 - Present

Tohoku University Hospital　University Hospital Pediatrics　Part-time Lecturer
2025/10 - Present

Iwate Prefectural Central Hospital　Pediatric Department

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Education 2

Tohoku University　Graduate School of Medicine

2015/04 - 2018/09
Tohoku University　Faculty of Medicine　School of Medicine

2002/04 - 2008/03

Professional Memberships 8

American Epilepsy Society

2020/11 - Present
日本てんかん学会
The International Child Neurology Association
American Society of Human Genetics
THE JAPANESE SOCIETY FOR PEDIATRIC INFECTIOUS DISEASES
JAPANESE SOCIETY OF EMERGENCY PEDIATRICS
The Japanese Society Of Child Neurology
JAPAN PEDIATRIC SOCIETY

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Research Interests 1

Hypoxic ischemic encephalopathy

Research Areas 1

Life sciences / Neurology /

Awards 5

太田敬三記念賞

2026/06　森永奉仕会　新生児低酸素性虚血性脳症の予後改善を目指して：ケトン体による神経細胞浮腫抑制効果と細胞内塩化物イオン調節
JUHN AND MARY WADA research encouragement award

2025/10　Japan Epilepsy Society　Role of NKCC1 and KCC2 during hypoxia-induced neuronal swelling in the neonatal neocortex
日本てんかん学会Sponsored Award（UCB賞）

2023/05　日本てんかん学会
成澤賞

2019/09　東北大学小児科　満期産脳性麻痺群の遺伝学的解析 -17例中9例で候補遺伝子変異を同定-
The 2nd JPS/SPR-Fostering leadership program award

2019/02　Japanese Pediatric Society

Papers 34

Solitary median maxillary central incisor syndrome caused by 22q11.2 microdeletion.

Hirohito Shima, Akinobu Miura, Sayaka Kawashima, Ikumi Umeki, Chisumi Sogi, Dai Suzuki, Yusuke Takezawa, Ryo Sato, Natsuko Arai-Ichinoi, Miki Kamimura, Ikuma Fujiwara, Mika Adachi, Aya Yamada, Hiroshi Kawame, Atsuo Kikuchi, Junko Kanno

Clinical pediatric endocrinology : case reports and clinical investigations : official journal of the Japanese Society for Pediatric Endocrinology　34　(1)　54-59　2025/01

DOI： 10.1297/cpe.2024-0024 　

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Solitary median maxillary central incisor (SMMCI) syndrome, the mildest form of the holoprosencephaly spectrum, is a rare anomaly characterized by the presence of a single midline central incisor in both the deciduous and permanent dentitions. Affected individuals can present with additional midline defects beyond dental findings. The 22q11.2 deletion syndrome (22q11.2 DS) arises from heterozygous microdeletions on chromosome 22q11.2, with breakpoints frequently located in eight clusters of low-copy repeats (LCR22A-H). Herein, we report an atypical case of 22q11.2 microdeletion in a male patient with SMMCI and additional features including hypothyroidism, ventricular septal defect, and several facial anomalies. The telomeric breakpoint was located in a segmental duplication 0.5 Mb distal to LCR22D, whereas the centromeric breakpoint was within LCR22C. Both segmental duplications shared a high level of sequence identity (97.2%), indicating the possibility of non-allelic homologous recombination (NAHR). This report supports the critical role of NAHR in the formation of rearrangements between regions other than LCR blocks and establishes a clinical association between 22q11.2 microdeletion and SMMCI.
脳性麻痺様症例の遺伝学的背景 91症例の病型別遺伝学的解析結果

竹澤祐介, 中村春彦, 西條直也, 相原悠, 堅田有宇, 及川善嗣, 佐藤亮, 大久保幸宗, 遠藤若葉, 阿部裕, 菊池敦生, 植松貢, 松本直通, 萩野谷和裕, 呉繁夫

脳と発達　56　(Suppl.)　S192-S192　2024/05
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
CACNA1D遺伝子バリアントを同定した難治てんかんの一例

宇根岡紗希, 後藤悠輔, 西條直也, 竹澤祐介, 植松有里佳, 植松貢, 浅見麻耶, 水間加奈子, 谷藤幸子, 赤坂真奈美, 高山順, 田宮元, 呉繁夫, 菊池敦生

脳と発達　56　(Suppl.)　S312-S312　2024/05
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
神経セロイドリポフスチン症2型(CLN2)との鑑別を有した発達性てんかん性脳症91型(DEE91)の1例

渋谷守栄, 中村晴彦, 西條直也, 宇根岡紗希, 竹澤祐介, 及川善嗣, 植松有里佳, 植松貢, 高山順, 呉繁雄, 田宮元, 菊池敦生

脳と発達　56　(Suppl.)　S313-S313　2024/05
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
Role of NKCC1 and KCC2 during hypoxia-induced neuronal swelling in the neonatal neocortex Peer-reviewed

Yusuke Takezawa, Rachel Langton, Samuel M. Baule, Miriam Bridget Zimmerman, Stephen Baek, Joseph Glykys

Neurobiology of Disease　178　106013-106013　2023/03
Publisher: Elsevier BV
DOI： 10.1016/j.nbd.2023.106013 　

ISSN： 0969-9961
Multiple Cerebral Hemorrhages and White Matter Lesions Developing after Severe hMPV Pneumonia in a Patient with Trisomy 13: A Case Report and Review of the Literature.

Moriei Shibuya, Noriko Togashi, Takehiko Inui, Yukimune Okubo, Wakaba Endo, Takuya Miyabayashi, Ryo Sato, Yusuke Takezawa, Kaori Kodama, Miki Ikeda, Aritomo Kawashima, Kazuhiro Haginoya

The Tohoku journal of experimental medicine　258　(1)　49-54　2022/08/06

DOI： 10.1620/tjem.2022.J056 　

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Human metapneumovirus (hMPV) is a common cause of upper and lower respiratory tract infections in children. A few case reports have described hMPV encephalitis or encephalopathy. Neuroimaging data on patients with hMPV encephalitis are scarce. We report a patient with trisomy 13 who developed severe hMPV pneumonia, multifocal cerebral and cerebellar hemorrhagic infarctions and extensive cerebral white matter demyelination. Although adult respiratory distress syndrome and disseminated intravascular coagulation contributed to the devastating central nervous system (CNS) lesions, endothelial dysfunction of the CNS caused by hMPV infection probably also played a pathophysiological role in this case.
A pediatric case of neuromyelitis optica spectrum disorder relapse with recurrent AQP4-IgG positivity

Kawashima, A., Hino-Fukuyo, N., Abe, Y., Takezawa, Y., Uematsu, M., Numata-Uematsu, Y., Takahashi, T., Nishiyama, S., Nakashima, I., Kure, S.

No to Hattatsu　53　(1)　2021

DOI： 10.11251/ojjscn.53.66 　

ISSN： 0029-0831
Primary pyomyositis in an infant following one-day afebrile upper limb monoplegia.

Yusuke Takezawa, Soh Niitsuma, Sayaka Kawashima, Shunsuke Miyano, Takashi Honma

The journal of medical investigation : JMI　68　(3.4)　372-375　2021

DOI： 10.2152/jmi.68.372 　

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Background : Pyomyositis is a subacute bacterial infection of the skeletal muscles. Its most common features are fever, muscle swelling, and focal pain. There have been insufficient data regarding pyomyositis in healthy infants. Case report : A one-month-old boy presented with an impairment of his left arm movement. He was well-nourished and not under any apparent distress. His vital signs were within the normal limits but neurological examination revealed left forearm paralysis. Physical examination showed no abnormal findings in the region from the left shoulder joint to the fingertips. Considering these factors, an intracranial pathology was initially suspected. However, he developed a fever, regular tachycardia, and swelling in the left forearm. Magnetic resonance imaging revealed inflammation in the left forearm muscles. He was diagnosed with bacterial myositis and started on intravenous antibiotics. On the 17th day, he was discharged with oral antibiotic treatment, which was completed over 25 days without any sequelae nor relapse. Conclusion : Here we report the case of Japanese primary pyomyositis following one-day afebrile upper limb monoplegia in an infant. Even when infants exhibit afebrile symptoms, a bacterial infection should be suspected. J. Med. Invest. 68 : 372-375, August, 2021.
抗AQP4抗体が再陽転化し再発した視神経脊髄炎関連疾患の小児例

川嶋有朋, 福與なおみ, 阿部裕, 竹澤祐介, 植松貢, 植松有里佳, 高橋利幸, 西山修平, 中島一郎, 呉繁夫

脳と発達　53　(1)　66-68　2021/01
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
リツキシマブを投与したCLIPPERSの1例

阿部裕, 遠藤若葉, 堅田有宇, 及川善嗣, 竹澤祐介, 植松有里佳, 植松貢, 呉繁夫

脳と発達　52　(Suppl.)　S267-S267　2020/08
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
特異な画像変化を呈し脳腫瘍の悪性転化を疑った限局性皮質形成異常の難治てんかん例

及川善嗣, 植松貢, 堅田有宇, 竹澤祐介, 植松有里佳, 阿部裕, 呉繁夫

脳と発達　52　(Suppl.)　S388-S388　2020/08
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
Correction: Biallelic GALM pathogenic variants cause a novel type of galactosemia. International-journal Peer-reviewed

Yoichi Wada, Atsuo Kikuchi, Natsuko Arai-Ichinoi, Osamu Sakamoto, Yusuke Takezawa, Shinya Iwasawa, Tetsuya Niihori, Hiromi Nyuzuki, Yoko Nakajima, Erika Ogawa, Mika Ishige, Hiroki Hirai, Hideo Sasai, Ryoji Fujiki, Matsuyuki Shirota, Ryo Funayama, Masayuki Yamamoto, Tetsuya Ito, Osamu Ohara, Keiko Nakayama, Yoko Aoki, Seizo Koshiba, Toshiyuki Fukao, Shigeo Kure

Genetics in medicine : official journal of the American College of Medical Genetics　22　(7)　1281-1281　2020/07

DOI： 10.1038/s41436-020-0836-z 　

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An amendment to this paper has been published and can be accessed via a link at the top of the paper.
Phenotype-genotype correlations in patients with GNB1 gene variants, including the first three reported Japanese patients to exhibit spastic diplegia, dyskinetic quadriplegia, and infantile spasms. International-journal Peer-reviewed

Wakaba Endo, Satoru Ikemoto, Noriko Togashi, Takuya Miyabayashi, Erika Nakajima, Shin-Ichiro Hamano, Moriei Shibuya, Ryo Sato, Yusuke Takezawa, Yukimune Okubo, Takehiko Inui, Mitsuhiro Kato, Toru Sengoku, Kazuhiro Ogata, Kohei Hamanaka, Takeshi Mizuguchi, Satoko Miyatake, Mitsuko Nakashima, Naomichi Matsumoto, Kazuhiro Haginoya

Brain & development　2019/11/14

DOI： 10.1016/j.braindev.2019.10.006 　

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We report the first three Japanese patients with missense variants in the GNB1 gene. Patients exhibited severe dyskinetic quadriplegia with cortical blindness and epileptic spasms, West syndrome (but with good outcomes), and hypotonic quadriplegia that later developed into spastic diplegia. Whole-exome sequencing revealed two recurrent GNB1 variants (p.Leu95Pro and p.Ile80Thr) and one novel variant (p.Ser74Leu). A recent investigation revealed large numbers of patients with GNB1 variants. Functional studies of such variants and genotype-phenotype correlation are required to enable future precision medicine.
GALMの両アレル性変異はガラクトース血症IV型を呈する

和田陽一, 菊池敦生, 市野井那津子, 坂本修, 岩澤伸哉, 竹澤祐介, 新堀哲也, 入月浩美, 中島葉子, 小川えりか, 石毛美夏, 平井洋生, 笹井英雄, 藤木亮次, 伊藤哲哉, 小原收, 青木洋子, 深尾敏幸, 呉繁夫

日本先天代謝異常学会雑誌　35　114-114　2019/09
Publisher: 日本先天代謝異常学会
ISSN： 0912-0122
Biallelic GALM pathogenic variants cause a novel type of galactosemia. International-journal Peer-reviewed

Yoichi Wada, Atsuo Kikuchi, Natsuko Arai-Ichinoi, Osamu Sakamoto, Yusuke Takezawa, Shinya Iwasawa, Tetsuya Niihori, Hiromi Nyuzuki, Yoko Nakajima, Erika Ogawa, Mika Ishige, Hiroki Hirai, Hideo Sasai, Ryoji Fujiki, Matsuyuki Shirota, Ryo Funayama, Masayuki Yamamoto, Tetsuya Ito, Osamu Ohara, Keiko Nakayama, Yoko Aoki, Seizo Koshiba, Toshiyuki Fukao, Shigeo Kure

Genetics in medicine : official journal of the American College of Medical Genetics　21　(6)　1286-1294　2019/06

DOI： 10.1038/s41436-018-0340-x 　

ISSN： 1098-3600

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PURPOSE: Galactosemia is caused by metabolic disturbances at various stages of galactose metabolism, including deficiencies in enzymes involved in the Leloir pathway (GALT, GALK1, and GALE). Nevertheless, the etiology of galactosemia has not been identified in a subset of patients. This study aimed to explore the causes of unexplained galactosemia. METHODS: Trio-based exome sequencing and/or Sanger sequencing was performed in eight patients with unexplained congenital galactosemia. In vitro enzymatic assays and immunoblot assays were performed to confirm the pathogenicity of the variants. RESULTS: The highest blood galactose levels observed in each patient were 17.3-41.9 mg/dl. Bilateral cataracts were observed in two patients. In all eight patients, we identified biallelic variants (p.Arg82*, p.Ile99Leufs*46, p.Gly142Arg, p.Arg267Gly, and p.Trp311*) in the GALM encoding galactose mutarotase, which catalyzes epimerization between β- and α-D-galactose in the first step of the Leloir pathway. GALM enzyme activities were undetectable in lymphoblastoid cell lines established from two patients. Immunoblot analysis showed the absence of the GALM protein in the patients' peripheral blood mononuclear cells. In vitro GALM expression and protein stability assays revealed altered stabilities of the variant GALM proteins. CONCLUSION: Biallelic GALM pathogenic variants cause galactosemia, suggesting the existence of type IV galactosemia.
小児期発症痙性対麻痺91症例の臨床的・遺伝学的解析

萩野谷和裕, 竹澤祐介, 乾健彦, 大久保幸宗, 佐藤亮, 冨樫紀子, 宮林拓矢, 渋谷守栄, 岩間一浩, 菊池敦生, 才津浩智, 松本直通, 呉繁夫

脳と発達　51　(Suppl.)　S294-S294　2019/05
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
Leucine-485 deletion variant of BRAF may exhibit the severe end of the clinical spectrum of CFC syndrome. International-journal Peer-reviewed

Suzuki-Muromoto S, Miyabayashi T, Nagai K, Yamamura-Suzuki S, Anzai M, Takezawa Y, Sato R, Okubo Y, Endo W, Inui T, Togashi N, Kikuchi A, Niihori T, Aoki Y, Kure S, Haginoya K

Journal of human genetics　64　(5)　499-504　2019/03

DOI： 10.1038/s10038-019-0579-3 　

ISSN： 1434-5161

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The genotype-phenotype correlation in BRAF variant in cardio-facio-cutaneous (CFC) syndrome is not clearly defined. Here we report a case with a severe clinical phenotype and a novel BRAF variant, p.Leu485del. The present case showed severe intellectual disability, impaired awareness, hyperekplexia, involuntary movements, early onset refractory seizures, and delayed myelination on brain magnetic resonance imaging as well as a polycystic and dysplastic kidney, which are previously unreported anomalies in CFC or RAS/mitogen-activated protein kinase syndromes related to BRAF variant. CFC syndrome, especially caused by BRAF variant, should be included in the differential diagnosis of patients with developmental and epileptic encephalopathies and hyperekplexia. Furthermore, we need to keep in mind that missense variants or the deletion of Leucine-485 may be associated with severe symptoms.
遺伝学的分析から特定された17患者中9名における病原性変異の候補(Genomic analysis identified candidate pathogenic variants in 9 of 17 patients)

Takezawa Yusuke, Kikuchi Atsuo, Haginoya Kazuhiro, Niihari Tetsuya, Numata-Uematsu Yurika, Inui Takehiko, Yamamura-Suzuki Saeko, Miyabayashi Takuya, Anzai Mai, Suzuki-Muromoto Sato, Okubo Yukimune, Endo Wakaba, Togashi Noriko, Kobayashi Yasuko, Onuma Akira, Funayama Ryo, Shirota Matsuyuki, Nakayama Keiko, Aoki Yoko, Kure Shigeo

日本小児科学会雑誌　123　(2)　292-292　2019/02
Publisher: (公社)日本小児科学会
ISSN： 0001-6543
Reply to: Avoid valproate in patients with IARS2 mutations Peer-reviewed

Takezawa, Y., Fujie, H., Kikuchi, A., Kure, S.

Brain and Development　41　(1)　122-122　2019/01
Publisher: Elsevier {BV}
DOI： 10.1016/j.braindev.2018.09.004 　

ISSN： 0387-7604 1872-7131
Novel IARS2 mutations in Japanese siblings with CAGSSS, Leigh, and West syndrome. International-journal Peer-reviewed

Yusuke Takezawa, Hiromi Fujie, Atsuo Kikuchi, Tetsuya Niihori, Ryo Funayama, Matsuyuki Shirota, Keiko Nakayama, Yoko Aoki, Masayuki Sasaki, Shigeo Kure

Brain & development　40　(10)　934-938　2018/11

DOI： 10.1016/j.braindev.2018.06.010 　

ISSN： 0387-7604

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BACKGROUND: IARS2 encodes isoleucine-tRNA synthetase, which is aclass-1 amino acyl-tRNA synthetase. IARS2 mutations are reported to cause Leigh syndrome or cataracts, growth hormone deficiency, sensory neuropathy, sensorineural hearing loss, and skeletal dysphasia syndrome (CAGSSS). To our knowledge, IARS2 mutations and diseases related to it have only been reported in three families. Here we report a case of two Japanese siblings with Leigh syndrome, some features of CAGSSS, and West syndrome that are found to have compound heterozygous novel IARS2 mutations. CASE REPORT: A 7-month-old Japanese girl presented with infantile spasms. Brain magnetic resonance imaging (MRI) revealed diffuse brain atrophy and hyperintensity in the bilateral basal ganglia. Three years later, her younger sister also presented with infantile spasms. MRI revealed diffuse brain atrophy and hyperintensity of the bilateral ganglia, suggesting Leigh syndrome. The siblings were identified with compound heterozygous missense mutations in IARS2, p.[(Phe227Ser)];[(Arg817His)]. CONCLUSION: This is the first case study reporting Leigh syndrome concomitant with some features of CAGSSS in siblings with novel IARS2 mutations, thereby broadening the phenotypic spectrum of IARS2-related disorders. Further studies are warranted to elucidate the nature of these disorders.
Reply to: A genomic cause of cerebral palsy should not change the clinical classification Peer-reviewed

Takezawa, Y., Kikuchi, A., Haginoya, K., Kure, S.

Annals of Clinical and Translational Neurology　5　(8)　1012-1012　2018/08

DOI： 10.1002/acn3.585 　

ISSN： 2328-9503
Rett-like features and cortical visual impairment in a Japanese patient with HECW2 mutation. International-journal Peer-reviewed

Haruhiko Nakamura, Mitsugu Uematsu, Yurika Numata-Uematsu, Yu Abe, Wakaba Endo, Atsuo Kikuchi, Yusuke Takezawa, Ryo Funayama, Matsuyuki Shirota, Keiko Nakayama, Tetsuya Niihori, Yoko Aoki, Kazuhiro Haginoya, Shigeo Kure

Brain & development　40　(5)　410-414　2018/05

DOI： 10.1016/j.braindev.2017.12.015 　

ISSN： 0387-7604

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Numerous genetic syndromes that include intellectual disability (ID) have been reported. Recently, HECW2 mutations were detected in patients with ID and growth development disorders. Four de novo missense mutations have been reported. Here, we report a Japanese girl with Rett-like symptoms of severe ID, hypotonia, refractory epilepsy, and stereotypical hand movement (hand tapping, flapping, and wringing) after the age of 1 year. Characteristically, she had cortical visual impairment. She had difficulty swallowing since the age of 4 years, and diminished activity was noticeable since the age of 12 years, suggesting neurodevelopmental regression. She has no acquired microcephaly, and brain magnetic resonance imaging showed non-specific mild cerebral and cerebellar atrophy without progression over time. Genetic analyses of MECP2, CDKL5, and FOXG1 were negative. Whole-exome sequencing analysis revealed a known de novo mutation (c.3988C > T) in HECW2. The characteristics of her clinical symptoms are severe cortical visual impairment and Rett-like phenotype such as involuntary movements and regression. This is the first report that patients with HECW2 mutation could show Rett-like feature.
Genomic analysis identifies masqueraders of full-term cerebral palsy. International-journal Peer-reviewed

Yusuke Takezawa, Atsuo Kikuchi, Kazuhiro Haginoya, Tetsuya Niihori, Yurika Numata-Uematsu, Takehiko Inui, Saeko Yamamura-Suzuki, Takuya Miyabayashi, Mai Anzai, Sato Suzuki-Muromoto, Yukimune Okubo, Wakaba Endo, Noriko Togashi, Yasuko Kobayashi, Akira Onuma, Ryo Funayama, Matsuyuki Shirota, Keiko Nakayama, Yoko Aoki, Shigeo Kure

Annals of clinical and translational neurology　5　(5)　538-551　2018/05

DOI： 10.1002/acn3.551 　

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Objective: Cerebral palsy is a common, heterogeneous neurodevelopmental disorder that causes movement and postural disabilities. Recent studies have suggested genetic diseases can be misdiagnosed as cerebral palsy. We hypothesized that two simple criteria, that is, full-term births and nonspecific brain MRI findings, are keys to extracting masqueraders among cerebral palsy cases due to the following: (1) preterm infants are susceptible to multiple environmental factors and therefore demonstrate an increased risk of cerebral palsy and (2) brain MRI assessment is essential for excluding environmental causes and other particular disorders. Methods: A total of 107 patients-all full-term births-without specific findings on brain MRI were identified among 897 patients diagnosed with cerebral palsy who were followed at our center. DNA samples were available for 17 of the 107 cases for trio whole-exome sequencing and array comparative genomic hybridization. We prioritized variants in genes known to be relevant in neurodevelopmental diseases and evaluated their pathogenicity according to the American College of Medical Genetics guidelines. Results: Pathogenic/likely pathogenic candidate variants were identified in 9 of 17 cases (52.9%) within eight genes: CTNNB1,CYP2U1,SPAST,GNAO1,CACNA1A,AMPD2,STXBP1, and SCN2A. Five identified variants had previously been reported. No pathogenic copy number variations were identified. The AMPD2 missense variant and the splice-site variants in CTNNB1 and AMPD2 were validated by in vitro functional experiments. Interpretation: The high rate of detecting causative genetic variants (52.9%) suggests that patients diagnosed with cerebral palsy in full-term births without specific MRI findings may include genetic diseases masquerading as cerebral palsy.
A patient with Muenke syndrome manifesting migrating neonatal seizures Peer-reviewed

Yukimune Okubo, Taro Kitamura, Mai Anzai, Wakaba Endo, Takehiko Inui, Yusuke Takezawa, Sato Suzuki-Muromoto, Takuya Miyabayashi, Noriko Togashi, Hiroshi Oba, Hirotomo Saitsu, Naomichi Matsumoto, Kazuhiro Haginoya

BRAIN & DEVELOPMENT　39　(10)　873-876　2017/11

DOI： 10.1016/j.braindev.2017.05.007 　

ISSN： 0387-7604

eISSN： 1872-7131
A novel mutation in the proteolytic domain of LONP1 causes atypical CODAS syndrome Peer-reviewed

Takehiko Inui, Mai Anzai, Yusuke Takezawa, Wakaba Endo, Yosuke Kakisaka, Atsuo Kikuchi, Akira Onuma, Shigeo Kure, Ichizo Nishino, Chihiro Ohba, Hirotomo Saitsu, Naomichi Matsumoto, Kazuhiro Haginoya

JOURNAL OF HUMAN GENETICS　62　(6)　653-655　2017/06

DOI： 10.1038/jhg.2017.11 　

ISSN： 1434-5161

eISSN： 1435-232X
無熱性の右上肢単麻痺を主訴に来院した乳児化膿性筋炎の一例

竹澤祐介, 新妻創, 宮野峻輔, 川嶋明香, 本間貴士, 伊藤健

日本小児科学会雑誌　121　(2)　350-350　2017/02
Publisher: (公社)日本小児科学会
ISSN： 0001-6543
First Japanese variant of late infantile neuronal ceroid lipofuscinosis caused by novel CLN6 mutations Peer-reviewed

Ryo Sato, Takehiko Inui, Wakaba Endo, Yukimune Okubo, Yusuke Takezawa, Mai Anzai, Hiroyuki Morita, Hirotomo Saitsu, Naomichi Matsumoto, Kazuhiro Haginoya

BRAIN & DEVELOPMENT　38　(9)　852-856　2016/10

DOI： 10.1016/j.braindev.2016.04.007 　

ISSN： 0387-7604

eISSN： 1872-7131
Patchy white matter hyperintensity in ring chromosome 18 syndrome Peer-reviewed

Mai Anzai, Natsuko Arai-Ichinoi, Yusuke Takezawa, Wakaba Endo, Takehiko Inui, Ryo Sato, Atsuo Kikuchi, Mitsugu Uematsu, Shigeo Kure, Kazuhiro Haginoya

PEDIATRICS INTERNATIONAL　58　(9)　919-922　2016/09

DOI： 10.1111/ped.13043 　

ISSN： 1328-8067

eISSN： 1442-200X
KCNQ2遺伝子変異を認めた早期乳児てんかん性脳症の2例

遠藤若葉, 竹澤祐介, 安西真衣, 乾健彦, 福與なおみ, 岩間一浩, 才津浩智, 松本直通, 萩野谷和裕

脳と発達　48　(Suppl.)　S270-S270　2016/05
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
Outcome of hemiplegic cerebral palsy born at term depends on its etiology Peer-reviewed

Yukihiro Kitai, Kazuhiro Haginoya, Satori Hirai, Kayo Ohmura, Kaeko Ogura, Takehiko Inui, Wakaba Endo, Yukimune Okubo, Mai Anzai, Yusuke Takezawa, Hiroshi Arai

BRAIN & DEVELOPMENT　38　(3)　267-273　2016/03

DOI： 10.1016/j.braindev.2015.09.007 　

ISSN： 0387-7604

eISSN： 1872-7131
FDG-PET study of patients with Leigh syndrome Peer-reviewed

Kauzhiro Haginoya, Tomohiro Kaneta, Noriko Togashi, Naomi Hino-Fukuyo, Tomoko Kobayashi, Mitsugu Uematsu, Taro Kitamura, Takehiko Inui, Yukimune Okubo, Yusuke Takezawa, Mai Anzai, Wakaba Endo, Noriko Miyake, Hirotomo Saitsu, Naomichi Matsumoto, Shigeo Kure

JOURNAL OF THE NEUROLOGICAL SCIENCES　362　309-313　2016/03

DOI： 10.1016/j.jns.2016.02.008 　

ISSN： 0022-510X

eISSN： 1878-5883
The efficacy of lamotrigine for atypical absence status epilepticus in a case of perioral myoclonia with absence Peer-reviewed

Yusuke Takezawa, Yosuke Kakisaka, Keisuke Wakusawa, Mamiko Ishitobi, Naomi Hino-Fukuyo, Takehiko Inui, Wakaba Endo, Mai Anzai, Nobukazu Nakasato, Kazuhiro Haginoya

Epilepsy and Seizure　8　(1)　1-8　2016
Publisher: Japan Epilepsy Society
DOI： 10.3805/eands.8.1 　

ISSN： 1882-5567

eISSN： 1882-5567
マイコプラズマ感染を契機として発症し、2回再発を来した抗NMDA抗体陽性辺縁系脳炎の9歳男児例

竹澤祐介, 植松貢, 新妻創, 宮野駿輔, 川嶋明香, 本間貴士, 伊藤健, 高橋幸利

脳と発達　47　(Suppl.)　S374-S374　2015/05
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
新生児重症Streptococcus bovis感染症の2例

竹澤祐介, 新妻創, 渡邉浩司, 今井香織, 三条雅敏, 高橋立子, 山田雅明

仙台赤十字病院医学雑誌　23　(1)　55-62　2014/05
Publisher: 仙台赤十字病院
ISSN： 0917-8724

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Misc. 13

101症例の頭部MRI画像(MRICS)別遺伝学的解析結果

竹澤祐介, 中村春彦, 西條直也, 相原悠, 堅田有宇, 及川善嗣, 佐藤亮, 大久保幸宗, 遠藤若葉, 阿部裕, 菊池敦生, 植松貢, 松本直通, 萩野谷和裕, 呉繁夫

脳と発達　57　(Suppl.)　s282-s282　2025/06
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
Dravet症候群へのフェンフルラミン投与について

植松貢, 竹澤祐介, 後藤悠輔, 宇根岡紗希, 児玉香織, 久保かほり, 植松有里佳, 菊池敦生

てんかん研究　42　(3)　2025

ISSN： 0912-0890
脳性麻痺様症例の遺伝学的背景 91症例の病型別遺伝学的解析結果

竹澤祐介, 中村春彦, 西條直也, 相原悠, 堅田有宇, 及川善嗣, 佐藤亮, 大久保幸宗, 遠藤若葉, 阿部裕, 菊池敦生, 植松貢, 松本直通, 萩野谷和裕, 呉繁夫

脳と発達　56　(Suppl.)　S192-S192　2024/05
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
眼球偏位・先天性筋緊張低下を認めた,チロシン水酸化酵素欠損症の1例

渋谷守栄, 佐藤亮, 宮林拓矢, 竹澤祐介, 遠藤若菜, 大久保幸宗, 乾健彦, 菊池敦生, 福與なおみ, 冨樫紀子, 秋山倫之, 萩野谷和裕

脳と発達　52　(1)　2020

ISSN： 0029-0831
満期産脳性麻痺群の網羅的遺伝学的解析 17例中9例で候補遺伝子変異を同定

竹澤祐介, 菊池敦生, 萩野谷和裕, 新堀哲也, 沼田有里佳, 松, 乾健彦, 山村菜絵子, 宮林拓矢, 安西真衣, 鈴木智, 室本, 大久保幸宗, 遠藤若葉, 冨樫紀子, 小林康子, 大沼晃, 舟山亮, 城田松之, 中山啓子, 青木洋子, 呉繁夫

日本小児科学会雑誌　123　(6)　1069-1070　2019/06
Publisher: (公社)日本小児科学会
ISSN： 0001-6543
GALMの両アレル性変異はガラクトース血症IV型を呈する(Blallelic GALM pathogenic variants cause a novel type of galactosemia)

和田陽一, 菊池敦生, 市野井那津子, 坂本修, 竹澤祐介, 岩澤伸哉, 新堀哲也, 入月浩美, 中島葉子, 小川えりか, 石毛美夏, 平井洋生, 笹井英雄, 藤木亮次, 伊藤哲也, 小原収, 青木洋子, 小柴生造, 深尾敏幸, 呉繁夫

日本小児科学会雑誌　123　(2)　280-280　2019/02
Publisher: (公社)日本小児科学会
ISSN： 0001-6543
GALMの両アレル性変異はガラクトース血症IV型を呈する(Blallelic GALM pathogenic variants cause a novel type of galactosemia)

和田陽一, 菊池敦生, 市野井那津子, 坂本修, 竹澤祐介, 岩澤伸哉, 新堀哲也, 入月浩美, 中島葉子, 小川えりか, 石毛美夏, 平井洋生, 笹井英雄, 藤木亮次, 伊藤哲也, 小原収, 青木洋子, 小柴生造, 深尾敏幸, 呉繁夫

日本小児科学会雑誌　123　(2)　280-280　2019/02
Publisher: (公社)日本小児科学会
ISSN： 0001-6543
22q11.2領域に欠失を認めた単発性正中上顎中切歯症候群の一例

菅野潤子, 竹澤祐介, 川嶋明香, 島彦仁, 曽木千純, 佐藤亮, 梅木郁美, 上村美季, 鈴木大, 菊池敦生, 川目裕, 藤原幾磨, 呉繁夫

日本小児科学会雑誌　123　(2)　401-401　2019/02
Publisher: (公社)日本小児科学会
ISSN： 0001-6543
22q11.2領域に欠失を認めた単発性正中上顎中切歯症候群の一例

菅野潤子, 竹澤祐介, 川嶋明香, 島彦仁, 曽木千純, 佐藤亮, 梅木郁美, 上村美季, 鈴木大, 菊池敦生, 川目裕, 呉繁夫, 藤原幾磨

日本内分泌学会雑誌　94　(2)　617-617　2018/09
Publisher: (一社)日本内分泌学会
ISSN： 0029-0661
てんかん性脳症と交互性片麻痺を呈しケトン食療法が有効であったCACNA1A遺伝子変異例

植松貢, 阿部裕, 遠藤若葉, 植松有里佳, 竹澤祐介, 菊池敦生, 呉繁夫

てんかん研究　36　(2)　452-452　2018/09
Publisher: (一社)日本てんかん学会
ISSN： 0912-0890

eISSN： 1347-5509
Rett症候群様の症状と皮質盲を伴うHECW2変異の1症例(Rett-like features and cortical visual impairment in a Japanese patient with HECW2 mutation)

中村春彦, 植松貢, 植松有里佳, 阿部裕, 遠藤若葉, 菊池敦夫, 竹澤祐介, 荻野谷和裕, 呉繁夫

脳と発達　50　(Suppl.)　S381-S381　2018/05
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

eISSN： 1884-7668
KCNA2変異によるてんかん性脳症の日本人初症例

相原悠, 植松貢, 及川善嗣, 竹澤祐介, 大久保幸宗, 植松有里佳, 菊池敦生, 呉繁夫

てんかん研究　35　(2)　440-440　2017/09
Publisher: (一社)日本てんかん学会
ISSN： 0912-0890
先天性多発性関節拘縮症に重度知的障害、進行性脳萎縮を来したZC4H2遺伝子変異を有する女児例

大久保幸宗, 遠藤若葉, 乾健彦, 竹澤祐介, 安西真衣, 佐藤亮, 市野井那津子, 菊池敦生, 植松貢, 呉繁夫, 萩野谷和裕

脳と発達　48　(Suppl.)　S418-S418　2016/05
Publisher: (一社)日本小児神経学会
ISSN： 0029-0831

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Presentations 35

満期産脳性麻痺群の網羅的遺伝学的解析 17例中9例で候補遺伝子変異を同定

竹澤祐介, 菊池敦生, 萩野谷和裕, 新堀哲也, 沼田有里佳, 松, 乾健彦, 山村菜絵子, 宮林拓矢, 安西真衣, 鈴木智, 室本, 大久保幸宗, 遠藤若葉, 冨樫紀子, 小林康子, 大沼晃, 舟山亮, 城田松之, 中山啓子, 青木洋子, 呉繁夫

日本小児科学会雑誌　2019/06
Thyrotropin-releasing hormone therapy is associated with favorable neurological outcomes after acute childhood encephalopathy: a retrospective study of 60 cases. International-presentation

Yusuke Takezawa, Masaru Takayanagi, Kazuhiro Haginoya

Pediatric Academic Societies Meeting　2019/04/30
22q11.2領域に欠失を認めた単発性正中上顎中切歯症候群の一例

菅野潤子, 竹澤祐介, 川嶋明香, 島彦仁, 曽木千純, 佐藤亮, 梅木郁美, 上村美季, 鈴木大, 菊池敦生, 川目裕, 藤原幾磨, 呉繁夫

日本小児科学会雑誌　2019/02
GALMの両アレル性変異はガラクトース血症IV型を呈する(Blallelic GALM pathogenic variants cause a novel type of galactosemia)

和田陽一, 菊池敦生, 市野井那津子, 坂本修, 竹澤祐介, 岩澤伸哉, 新堀哲也, 入月浩美, 中島葉子, 小川えりか, 石毛美夏, 平井洋生, 笹井英雄, 藤木亮次, 伊藤哲也, 小原収, 青木洋子, 小柴生造, 深尾敏幸, 呉繁夫

日本小児科学会雑誌　2019/02
Genomic analysis identifies pathogenic variants in 9 of 17 cases with full-term cerebral palsy International-presentation

Takezawa Y, Kikuchi A, Haginoya K, Niihori T, Numata‐Uematsu Y, Inui T, Yamamura‐Suzuki S, Miyabayashi T, Anzai M, Suzuki‐Muromoto S, Okubo Y, Endo W, Togashi N, Kobayashi Y, Onuma A, Funayama R, Shirota M, Nakayama K, Aoki Y, Kure S

International Child Neurology Congress Mumbai 2018.　2018/12/16
Genetic background of unexplained full-term cerebral palsy: Genomic analysis identifies candidate pathogenic variants in 9 of 17 patients. International-presentation

Takezawa Y, Kikuchi A, Haginoya K, Niihori T, Numata‐Uematsu Y, Inui T, Yamamura‐Suzuki S, Miyabayashi T, Anzai M, Suzuki‐Muromoto S, Okubo Y, Endo W, Togashi N, Kobayashi Y, Onuma A, Funayama R, Shirota M, Nakayama K, Aoki Y, Kure S

American Society of Human Genetics 2018 annual meeting.　2018/10/19
てんかん性脳症と交互性片麻痺を呈しケトン食療法が有効であったCACNA1A遺伝子変異例

植松貢, 阿部裕, 遠藤若葉, 植松有里佳, 竹澤祐介, 菊池敦生, 呉繁夫

てんかん研究　2018/09
22q11.2領域に欠失を認めた単発性正中上顎中切歯症候群の一例

菅野潤子, 竹澤祐介, 川嶋明香, 島彦仁, 曽木千純, 佐藤亮, 梅木郁美, 上村美季, 鈴木大, 菊池敦生, 川目裕, 呉繁夫, 藤原幾磨

日本内分泌学会雑誌　2018/09
Rett症候群様の症状と皮質盲を伴うHECW2変異の1症例(Rett-like features and cortical visual impairment in a Japanese patient with HECW2 mutation)

中村春彦, 植松貢, 植松有里佳, 阿部裕, 遠藤若葉, 菊池敦夫, 竹澤祐介, 荻野谷和裕, 呉繁夫

脳と発達　2018/05
Genomic analysis identifies masqueraders of full-term cerebral palsy. International-presentation Invited

Takezawa Y, Haginoya K

The 2nd International Cerebral Palsy Genomics Consortium Conference　2018/04/14
KCNA2変異によるてんかん性脳症の日本人初症例

相原悠, 植松貢, 及川善嗣, 竹澤祐介, 大久保幸宗, 植松有里佳, 菊池敦生, 呉繁夫

てんかん研究　2017/09
Factors associated with long-term neurological outcomes after childhood acute encephalopathy: A retrospective study of 74 cases. International-presentation

Takezawa Y, Anzai M, Endo W, Inui T, Kitamura T, Takayanagi M, Shirai I, Haginoya K

The 14th Asian and Oceanian Congress of Child Neurology.　2017/05/13
本邦初のCLN6遺伝子変異による後期乳児型神経セロイドリポフスシノーシスの一例

佐藤亮, 乾健彦, 遠藤若葉, 大久保幸宗, 竹澤祐介, 安西真衣, 森田浩之, 才津浩智, 松本直道, 萩野谷和裕

脳と発達　2017/05
PCDH12遺伝子に新規複合ヘテロ変異を同定した男児例

鈴木智, 宮林拓矢, 竹澤祐介, 大久保幸宗, 遠藤若葉, 乾健彦, 涌沢圭介, 冨樫紀子, 萩野谷和裕, 中島光子, 才津浩智, 松本直通

脳と発達　2017/05
FOXG1遺伝子の欠失が判明した後天性小頭症、てんかん、movement disorderの一例

遠藤若葉, 竹澤祐介, 大久保幸宗, 乾健彦, 鈴木智, 宮林拓矢, 冨樫紀子, 萩野谷和裕

脳と発達　2017/05
急性脳症による後遺症を呈した74症例のGross motor function classification system(GMFCS)の後方視的検討

竹澤祐介, 安西真衣, 遠藤若葉, 乾健彦, 白井育子, 萩野谷和裕

日本小児科学会雑誌　2017/05
FOXG1の欠失が判明した後天性小頭症、てんかん、movement disorderの1例

遠藤若葉, 竹澤祐介, 冨樫紀子, 乾健彦, 大久保幸宗, 鈴木智, 宮林拓矢, 山村菜絵子, 才津浩智, 松本直通, 萩野谷和裕

脳と発達　2017/03
無熱性の右上肢単麻痺を主訴に来院した乳児化膿性筋炎の一例

竹澤祐介, 新妻創, 宮野峻輔, 川嶋明香, 本間貴士, 伊藤健

日本小児科学会雑誌　2017/02
Factors associated with intractable epilepsy after childhood acute encephalopathy: A retrospective study of 74 cases. International-presentation

Takezawa Y, Anzai M, Endo W, Inui T, Kitamura T, Takayanagi M, Shirai I, Haginoya K

The 18th Annual Meeting of Infantile Seizure Society ISAE 2016.　2016/07/03
先天性多発性関節拘縮症に重度知的障害、進行性脳萎縮を来したZC4H2遺伝子変異を有する女児例

大久保幸宗, 遠藤若葉, 乾健彦, 竹澤祐介, 安西真衣, 佐藤亮, 市野井那津子, 菊池敦生, 植松貢, 呉繁夫, 萩野谷和裕

脳と発達　2016/05
先天性内反足を伴う運動感覚性ニューロパチー6例の検討

安西真衣, 佐藤亮, 竹澤祐介, 遠藤若葉, 乾健彦, 田中佳子, 萩野谷和裕

脳と発達　2016/05
末梢神経症状と錐体路症状を併せ持つ疾患群の原因は多様である

萩野谷和裕, 乾健彦, 遠藤若葉, 安西真衣, 竹澤祐介, 才津浩智

脳と発達　2016/05
KCNQ2遺伝子変異を認めた早期乳児てんかん性脳症の2例

遠藤若葉, 竹澤祐介, 安西真衣, 乾健彦, 福與なおみ, 岩間一浩, 才津浩智, 松本直通, 萩野谷和裕

脳と発達　2016/05
退行を来したCODAS症候群の1例

乾健彦, 安西真衣, 竹澤祐介, 遠藤若葉, 涌澤圭介, 田中総一郎, 大場ちひろ, 才津浩智, 松本直通, 萩野谷和裕

脳と発達　2016/05
当センターにおける急性脳症後遺症の70例の長期予後とリハビリテーション

竹澤祐介, 安西真衣, 遠藤若葉, 乾健彦, 白井育子, 萩野谷和裕

脳と発達　2016/05
マイコプラズマ感染を契機として発症し、2回再発を来した抗NMDA抗体陽性辺縁系脳炎の9歳男児例

竹澤祐介, 植松貢, 新妻創, 宮野駿輔, 川嶋明香, 本間貴士, 伊藤健, 高橋幸利

脳と発達　2015/05
ムンプスワクチン接種後に発症した可逆性脳梁膨大部病変を有する軽症脳炎・脳症の1例

佐々木和人, 内田崇, 高柳勝, 中村洋心, 高橋俊成, 佐藤大記, 竹澤祐介, 楠本耕平, 鈴木力生, 北村太郎, 西尾利之, 西村秀一, 石井清, 大浦敏博

仙台市立病院医学雑誌　2014/08

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2歳11ヵ月男児。ムンプスワクチン(星野株)接種後21日後に発熱、24日後に構音障害が出現した。臨床経過、各所見よりムンプスワクチン接種による無菌性髄膜炎が疑われた。MRIでは、脳梁膨大部の拡散強調像で高信号域、apparent diffusion coefficient低下、脳波は睡眠紡錘波が認められた。D-mannitol、aciclovir、cefotaxime、dexamethasoneで加療したところ解熱し、第12病日に構音障害や画像所見は改善し入院時検査で陰性であった血清ムンプスIgM、IgGは陽性となった。また、髄液検査でムンプスウイルス(星野株)を検出したことより、ムンプスワクチンによる可逆性脳梁膨大部病変を有する軽症脳炎・脳症(MERS)と確定診断した。全身状態は良好で神経学的後遺症はなく、第13病日に独歩にて退院した。
ムンプスワクチン接種後に発症した可逆性脳梁膨大部病変を有する軽症脳炎・脳症の1例

佐々木和人, 内田崇, 高柳勝, 中村洋心, 高橋俊成, 佐藤大記, 竹澤祐介, 楠本耕平, 鈴木力生, 北村太郎, 西尾利之, 大浦敏博, 西村秀一

日本小児科学会雑誌　2014/05
宮城県内の小児重症患者搬送体制への提言

中村洋心, 楠本耕平, 鈴木力生, 村田祐二, 佐々木和人, 内田崇, 高橋俊成, 佐藤大記, 竹澤祐介, 北村太郎, 西尾利之, 高柳勝, 大浦敏博, 角田文彦, 大久田隆, 稲垣徹史, 虻川大樹

日本小児科学会雑誌　2014/05
けいれん重積発作を呈したパラインフルエンザウイルス3型感染症の検討

高橋俊成, 佐藤大記, 高柳勝, 中村洋心, 佐々木和人, 内田崇, 竹澤祐介, 楠本耕平, 鈴木力生, 北村太郎, 西尾利之, 村田祐二, 大浦敏博, 石井清, 高橋雅司, 西村秀一

小児感染免疫　2014/04
当センターで経験した点状軟骨異形成症の環軸椎亜脱臼に関する臨床的経過の検討

竹澤祐介, 余谷暢之, 石黒精, 紙谷万里子, 益田博司, 師田信人, 小崎里華, 宮嵜治, 西村玄, 阪井裕一

日本小児科学会雑誌　2013/02
マクロファージ活性化症候群を発症した慢性肉芽腫症の3例

村山静子, 井田博幸, 明城和子, 竹澤祐介, 前川貴伸, 石黒精, 大石勉, 河合利尚

小児感染免疫　2012/04
小児期胃腸炎における高尿酸血症の検討

竹澤祐介, 田中康子, 宮城なつき, 明城和子, 堀内清華, 小林由典, 土田尚, 中舘尚也, 石黒精, 阪井裕一

日本小児科学会雑誌　2012/02
マクロファージ活性化症候群を合併した慢性肉芽腫症の1例

竹澤祐介, 生田泰久, 長野智那, 川口隆弘, 前川貴伸, 石黒精, 阪井裕一, 河合利尚

日本小児科学会雑誌　2011/11
急性骨髄性白血病に対する化学療法後の好中球減少症においてMRSA肝脾膿瘍を合併した1例

竹澤祐介, 関正則, 高川真徳

臨床血液　2009/08

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Research Projects 4

新生児低酸素性虚血性脳症におけるNa/Clイオン流入抑制は神経細胞浮腫を軽減する

竹澤祐介

Offer Organization: 日本学術振興会

System: 科学研究費助成事業

Category: 若手研究

Institution: 東北大学

2025/04 - 2027/03
Analysis of predictors of attention deficit / hyperactivity disorder by longitudinal analysis

Offer Organization: Japan Society for the Promotion of Science

System: Grants-in-Aid for Scientific Research

Category: Grant-in-Aid for Scientific Research (B)

Institution: Tokai Gakuen University

2020/04/01 - 2023/03/31
Calcium overload and cell swelling during hypoxia in the neonatal brain

Offer Organization: American Epilepsy Society

System: Postdoctoral Research Fellowships

Institution: The University of Iowa, Iowa Neuroscience Institute

2021/07 - 2022/06
Identifying genetic background of full-term cerebral palsy based on brain MRI findings. Competitive

Takezawa Yusuke

Offer Organization: Japan Society for the Promotion of Science

System: Grants-in-Aid for Scientific Research(KAKEN)

Category: Grant-in-Aid for Early-Career Scientists

Institution: Tohoku University

2019/04 - 2022/04