Details of the Researcher

PHOTO

Hiroyuki Igarashi
Section
Graduate School of Pharmaceutical Sciences
Job title
Assistant Professor
Degree
Profile

東北大学医学系研究科にて修士, 博士課程を修了
八尾寛教授の下で
1) 新規オプトジェネティクスツールの開発（オルガネラ・オプトジェネティクス）
2) コネクトミクス/オプトジェネティクス研究に資する新規トランスジェニック系統の開発
3) 多能性間葉系幹細胞であるMuse細胞の分化メカニズム解明に向けた研究
に取り組んだ。

博士号取得後は、カナダ・西オンタリオ大学にてストレス応答の神経基盤研究を行った。

この際、日本学術振興会 若手研究者海外挑戦プログラムの支援を受けて留学を開始し、後にBrainsCAN Fellowship、日本学術振興会 海外特別研究員に採用された。

2023年6月より現職。全身性ストレス応答の起点となるCRFニューロンに着目した網羅的計測法を確立し、CRFニューロンの入出力制御機構に着目したストレス適応メカニズムの解明を目指す。

【個人HP】

https://sites.google.com/view/igarashi-group

Research History 11

  • 2026/04 - Present
    Niigata University　Faculty of Medicine　Adjunct lecturer

  • 2026/04 - Present
    Memorial University of Newfoundland　Division of BioMedical Sciences, Faculty of Medicine　Adjunct Professor

  • 2023/06 - Present
    Tohoku University　Graduate School of Pharmaceutical Sciences　Assistant Professor

  • 2021/01 - 2023/05
    Japan Society for the Promotion of Science　overseas research fellow

  • 2018/09 - 2020/12
    BrainsCAN Postdoctoral Associate

  • 2018/03 - 2019/02
    日本学術振興会 平成29年度 若手研究者海外挑戦プログラム

  • 2016/04 - 2018/03
    東北大学学際高等研究教育院 博士研究教育院生

  • 2016/04 - 2018/03
    日本学術振興会 特別研究員(DC1)

  • 2012/04 - 2018/03
    神経細胞制御学分野 指導教員：八尾 寛

  • 2015/04 - 2016/03
    本庄国際奨学財団 日本人大学院奨学生

  • 2014/04 - 2015/03
    東北大学国際高等研究教育院 修士研究教育院生

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Professional Memberships 4

  • The Japanese Pharmacological Society

    2025/02 - Present

  • Japan Neuroendocrine Society

    2023/06 - Present

  • THE JAPAN NEUROSCIENCE SOCIETY

  • PHYSIOLOGICAL SOCIETY OF JAPAN

Research Interests 2

  • 神経内分泌

  • ムーンショットJIZAI20250プロジェクト

Research Areas 2

  • Life sciences / Physiology /

  • Life sciences / Neuroscience - general /

Awards 3

  1. 第23回日本神経内分泌学会 若手研究奨励賞

    2024/10　日本神経内分泌学会　コルチコトロピン放出ホルモンの生体内放出パターンの理解と生理学的意義の解明

  2. 令和5年度コニカミノルタ画像科学奨励賞 優秀賞

    2024/01　コニカミノルタ科学技術振興財団　嗅ぎ分け細胞によるストレスホルモンのリアルタイム蛍光検出と応用

  3. 第2 回筋学会学術集会 Student’s Awards 最優秀賞

    2016/08　日本筋学会

Papers 17

  1. Stressor-specific dynamic patterns of noradrenaline release in the paraventricular nucleus of the hypothalamus in freely moving mice

    Ryunosuke Ono, Keiichi Itoi, Kenji Sakimura, Takuya Sasaki, Hiroyuki Igarashi

    2026/07/28

    DOI： 10.64898/2026.07.25.740743 　

  2. A framework for rapid three-dimensional visualization of whole cerebral vasculature in mice using high-frequency ultrafast ultrasound with continuous mechanical scanning and ensemble size optimization

    Yoshihisa Kaneko, Hiroyuki Igarashi, Haruya Yagishita, Anam Bhatti, Takuya Sasaki, Yoshifumi Saijo, Takuro Ishii

    Japanese Journal of Applied Physics　65　(6)　06SP04-06SP04　2026/03/18

    Publisher: IOP Publishing

    DOI： 10.35848/1347-4065/ae4b46 　

    ISSN： 0021-4922

    eISSN： 1347-4065

    More details Close

    Abstract Understanding cerebrovascular dynamics requires imaging techniques that provide high spatial and temporal resolution over a wide field of view in small animals. In this study, we developed a high-frequency ultrafast ultrasound framework that combines contrast-enhanced imaging with continuous mechanical scanning to achieve volumetric acquisition of whole-brain in mice within 5 s. The acquired data were processed using section-wise singular value decomposition, in which the in-phase and quadrature dataset is divided into spatial sections according to the elevational resolution of the transducer. This approach enabled robust extraction of blood-flow signals, preserved spatial continuity across all three dimensions, and balanced Doppler sensitivity with elevation resolution. The reconstructed power Doppler images successfully visualized major cerebral arteries and veins, branching vessels, and fine cortical and subcortical microvasculature. These results demonstrate the feasibility of rapid three-dimensional visualization of whole-brain vascular networks, providing a foundation for future studies requiring fast and repeated monitoring of cerebrovascular dynamics.

  3. Second-Scale Neural Dynamics Shape Hormonal Outputs in Hypothalamic CRH Neurons

    Hiroyuki Igarashi, Samuel Mestern, Aoi Ichiyama, Susheel Vijayraghavan, Julio C Martinez-Trujillo, Rithwik Ramachandran, Wataru Inoue

    2025/03

    DOI： 10.1101/2025.03.18.641872 　

  4. THREE-DIMENSIONAL IMAGING OF RODENT BRAIN VASCULATURE USING ULTRAFAST HIGH- FREQUENCY ULTRASOUND WITH CONTINUOUS MECHANICAL SCANNING

    Yoshihisa KANEKO, Haruya Yagishita, Anam Bhatti, Yoshifumi Saijo, Hiroyuki Igarashi, Takuro Ishii

    Ultrasound in Medicine & Biology　51　S117-S117　2025

    Publisher: Elsevier BV

    DOI： 10.1016/j.ultrasmedbio.2025.11.132 　

    ISSN： 0301-5629

  5. Spike frequency adaptation in primate lateral prefrontal cortex neurons results from interplay between intrinsic properties and circuit dynamics

    Nils A. Koch, Benjamin W. Corrigan, Michael Feyerabend, Roberto A. Gulli, Michelle S. Jimenez-Sosa, Mohamad Abbass, Julia K. Sunstrum, Sara Matovic, Megan Roussy, Rogelio Luna, Samuel A. Mestern, Borna Mahmoudian, Susheel Vijayraghavan, Hiroyuki Igarashi, Kartik S. Pradeepan, William J. Assis, J. Andrew Pruszynski, Shreejoy Tripathy, Jochen F. Staiger, Guillermo Gonzalez-Burgos, Andreas Neef, Stefan Treue, Stefan Everling, Wataru Inoue, Anmar Khadra, Julio C. Martinez-Trujillo

    Cell Reports　44　(1)　115159-115159　2025/01

    Publisher: Elsevier BV

    DOI： 10.1016/j.celrep.2024.115159 　

    ISSN： 2211-1247

  6. Direct modulation of CRH nerve terminal function by noradrenaline and corticosterone

    Emmet M. Power, Dharshini Ganeshan, Jamieson Paul, Hiroyuki Igarashi, Wataru Inoue, Karl J. Iremonger

    The Journal of Neuroscience　e1092242024-e1092242024　2024/12/05

    Publisher: Society for Neuroscience

    DOI： 10.1523/jneurosci.1092-24.2024 　

    ISSN： 0270-6474

    eISSN： 1529-2401

    More details Close

    Nerve terminals are the final point of regulation before neurosecretion. As such, neuromodulators acting on nerve terminals can exert significant influence on neural signalling. Hypothalamic corticotropin-releasing hormone (CRH) neurons send axonal projections to the median eminence where CRH is secreted to stimulate the hypothalamic-pituitary-adrenal (HPA) axis. Noradrenaline and corticosterone are two of the most important neuromodulators of HPA axis function; noradrenaline excites CRH neurons and corticosterone inhibits CRH neurons by negative feedback. Here, we used GCaMP6f Ca2+imaging and measurement of nerve terminal CRH secretion using sniffer cells to determine whether these neuromodulators act directly on CRH nerve terminals in male mice. Contrary to expectations, noradrenaline inhibited action potential-dependent Ca2+elevations in CRH nerve terminals and suppressed evoked CRH secretion. This inhibitory effect was blocked by α2-adrenoreceptor antagonism. Corticosterone also suppressed evoked CRH peptide secretion from nerve terminals, independent of action potential-dependent Ca2+levels. This inhibition was prevented by the glucocorticoid receptor antagonist, RU486, and indicates that CRH nerve terminals may be a site of fast glucocorticoid negative feedback. Together these findings establish median eminence nerve terminals as a key site for regulation of the HPA axis. Significance StatementCorticotropin-releasing hormone (CRH) neurons control the stress axis. Noradrenaline and corticosterone are two signalling molecules that control CRH neuron cell body excitability. However, their effect on CRH nerve terminal function is unknown. To examine this, we performed live Ca2+imaging and measured CRH secretion. We found that noradrenaline suppressed nerve terminal Ca2+levels and inhibited nerve terminal CRH secretion. Corticosterone had no effect on nerve terminal Ca2+, but inhibited nerve terminal CRH secretion. This suggests that CRH nerve terminals may be a site of fast corticosteroid negative feedback. Together, these data demonstrate that CRH nerve terminals are a critical point of regulation in the control of the stress axis.

  7. Spike frequency adaptation in primate lateral prefrontal cortex neurons results from interplay between intrinsic properties and circuit dynamics

    Nils A. Koch, Benjamin W. Corrigan, Michael Feyerabend, Roberto A. Gulli, Michelle S. Jimenez-Sosa, Mohamad Abbass, Julia K. Sunstrum, Sara Matovic, Megan Roussy, Rogelio Luna, Samuel A. Mestern, Borna Mahmoudian, Susheel Vijayraghavan, Hiroyuki Igarashi, Kartik S. Pradeepan, William J. Assis, J. Andrew Pruszynski, Shreejoy Tripathy, Jochen F. Staiger, Guillermo Gonzalez-Burgos, Andreas Neef, Stefan Treue, Stefan Everling, Wataru Inoue, Anmar Khadra, Julio C. Martinez-Trujillo

    bioRxiv　2024/09/05

    Publisher: Cold Spring Harbor Laboratory

    DOI： 10.1101/2024.09.03.610998 　

    More details Close

    Abstract Recordings of cortical neurons isolated from brain slices and dissociated from their networks, display intrinsic spike frequency adaptation (I-SFA) to a constant current input. Interestingly, extracellular recordings in behaving subjects also show extrinsic-SFA (E-SFA) in response to sustained visual stimulation. Because neurons are isolated from brain networks in slice recordings, it is challenging to infer how I-SFA contributes to E-SFA in interconnected brains during behavior. To investigate this, we recorded responses of macaque lateral prefrontal cortex neuronsin vivoduring a visually guided saccade task and in acute brain slicesin vitro. Broad spiking (BS) putative pyramidal cells and narrow spiking (NS) putative inhibitory interneurons exhibited E-SFAin vivo. In acute brain slices, both cell types displayed I-SFA though their magnitudes differed. To investigate howin vitroI-SFA contributes toin vivoE-SFA, we developed a data-driven hybrid circuit model in which local NS neurons are driven by BS input. We observed that model NS cell responses show longer SFA than observedin vivo. Introducing inhibition of NS cells to the model circuit removed this discrepancy. Our results indicate that both I-SFA and inhibitory circuit dynamics contribute to E-SFA in LPFC neurons. They highlight the contribution of single neuron and network dependent computations to neural activity underlying behavior.

  8. Direct modulation of CRH nerve terminal function by noradrenaline and corticosterone

    Emmet M. Power, Dharshini Ganeshan, Jamieson Paul, Hiroyuki Igarashi, Wataru Inoue, Karl J. Iremonger

    bioRxiv　2024/06/12

    Publisher: Cold Spring Harbor Laboratory

    DOI： 10.1101/2024.06.11.598540 　

    More details Close

    Abstract Nerve terminals are the final point of regulation before neurosecretion. As such, neuromodulators acting on nerve terminals can exert significant influence on neural signalling. Hypothalamic corticotropin-releasing hormone (CRH) neurons send axonal projections to the median eminence where CRH is secreted to stimulate the hypothalamic-pituitary-adrenal (HPA) axis. Noradrenaline and corticosterone are two of the most important neuromodulators of HPA axis function; noradrenaline excites CRH neurons and corticosterone inhibits CRH neurons by negative feedback. Here, we used GCaMP6f Ca2+imaging and measurement of nerve terminal CRH secretion using sniffer cells to determine whether these neuromodulators act directly on CRH nerve terminals. Contrary to expectations, noradrenaline inhibited action potential-dependent Ca2+elevations in CRH nerve terminals and suppressed evoked CRH secretion. This inhibitory effect was blocked by α2-adrenoreceptor antagonism. Corticosterone also suppressed evoked CRH peptide secretion from nerve terminals, independent of action potential-dependent Ca2+levels. This inhibition was prevented by the glucocorticoid receptor antagonist, RU486, and indicates that CRH nerve terminals may be a site of fast glucocorticoid negative feedback. Together these findings establish median eminence nerve terminals as a key site for regulation of the HPA axis. Significance Corticotropin-releasing hormone (CRH) neurons control the stress axis. Noradrenaline and corticosterone are two signalling molecules that control CRH neuron cell body excitability. However, their effect on CRH nerve terminal function is unknown. To examine this, we performed live Ca2+imaging and measured CRH secretion. We found that noradrenaline suppressed nerve terminal Ca2+levels and inhibited nerve terminal CRH secretion. Corticosterone had no effect on nerve terminal Ca2+, but inhibited nerve terminal CRH secretion. This suggests that CRH nerve terminals may be a site of fast corticosteroid negative feedback. Together, these data demonstrate that CRH nerve terminals are a critical point of regulation in the control of the stress axis.

  9. 実験動物としてのラットの有用性 ラットを用いたオプトジェネティクス(光遺伝学)

    八尾 寛, 深澤 有吾, 冨田 浩史, 五十嵐 敬幸

    LABIO 21　(80)　5-8　2020/05

    Publisher: (公社)日本実験動物協会

    ISSN： 1345-9147

  10. Neuronal Hypertrophy Dampens Neuronal Intrinsic Excitability and Stress Responsiveness During Chronic Stress Peer-reviewed

    Sara Matovic, Aoi Ichiyama, Hiroyuki Igarashi, Eric W Salter, Julia K Sunstrum, Xue Fan Wang, Mathilde Henry, Eric S Kuebler, Nathalie Vernoux, Julio Martinez-Trujillo, Marie-Eve Tremblay, Wataru Inoue

    The journal of physiology　2020/04/29

  11. Optogenetic analysis of respiratory neuronal networks in the ventral medulla of neonatal rats producing channelrhodopsin in Phox2b-positive cells Peer-reviewed

    Keiko Ikeda, Hiroyuki Igarashi, Hiromu Yawo, Kazuto Kobayashi, Satoru Arata, Kiyoshi Kawakami, Masahiko Izumizaki, Hiroshi Onimaru

    Pflügers Archiv - European Journal of Physiology　471　(11-12)　1419-1439　2019/12

    Publisher: Springer Science and Business Media LLC

    DOI： 10.1007/s00424-019-02317-9 　

    ISSN： 0031-6768

    eISSN： 1432-2013

  12. Responses to hypercapnia and hypoxia of neurons in the cardio-respiratory center of the ventral medulla of newborn rats

    Hiroshi Onimaru, Keiko Ikeda, Hiroyuki Igarashi, Hiromu Yawo, Kazuto Kobayashi, Satoru Arata, Kiyoshi Kawakami, Masahiko Izumizaki

    IBRO Reports　6　S409-S409　2019/09

    Publisher: Elsevier BV

    DOI： 10.1016/j.ibror.2019.07.1303 　

    ISSN： 2451-8301

  13. Targeted expression of step-function opsins in transgenic rats for optogenetic studies Peer-reviewed

    Hiroyuki Igarashi, Keiko Ikeda, Hiroshi Onimaru, Ryosuke Kaneko, Kyo Koizumi, Kaoru Beppu, Kayo Nishizawa, Yukari Takahashi, Fusao Kato, Ko Matsui, Kazuto Kobayashi, Yuchio Yanagawa, Shin-Ichi Muramatsu, Toru Ishizuka, Hiromu Yawo

    Scientific Reports　8　(1)　5435　2018/12/01

    Publisher: Nature Publishing Group

    DOI： 10.1038/s41598-018-23810-8 　

    ISSN： 2045-2322

  14. Organelle Optogenetics: Direct Manipulation of Intracellular Ca2+ Dynamics by Light International-journal Peer-reviewed

    Asano T, Igarashi H, Ishizuka T, Yawo H

    Front. Neurosci.　12　(561)　561-561　2018/08

    DOI： 10.3389/fnins.2018.00561 　

    More details Close

    As one of the ubiquitous second messengers, the intracellular Ca2+, has been revealed to be a pivotal regulator of various cellular functions. Two major sources are involved in the initiation of Ca2+-dependent signals: influx from the extracellular space and release from the intracellular Ca2+ stores such as the endoplasmic/sarcoplasmic reticulum (ER/SR). To manipulate the Ca2+ release from the stores under high spatiotemporal precision, we established a new method termed "organelle optogenetics." That is, one of the light-sensitive cation channels (channelrhodopsin-green receiver, ChRGR), which is Ca2+-permeable, was specifically targeted to the ER/SR. The expression specificity as well as the functional operation of the ER/SR-targeted ChRGR (ChRGRER) was evaluated using mouse skeletal myoblasts (C2C12): (1) the ChRGRER co-localized with the ER-marker KDEL; (2) no membrane current was generated by light under whole-cell clamp of cells expressing ChRGRER; (3) an increase of fluorometric Ca2+ was evoked by the optical stimulation (OS) in the cells expressing ChRGRER in a manner independent on the extracellular Ca2+ concentration ([Ca2+]o); (4) the ΔF/F0 was sensitive to the inhibitor of sarco/endoplasmic reticulum Ca2+-ATPase (SERCA) and (5) the store-operated Ca2+ entry (SOCE) was induced by the OS in the ChRGRER-expressing cells. Our organelle optogenetics effectively manipulated the ER/SR to release Ca2+ from intracellular stores. The use of organelle optogenetics would reveal the neuroscientific significance of intracellular Ca2+ dynamics under spatiotemporal precision.

  15. A Chimera Na+ -Pump Rhodopsin as an Effective Optogenetic Silencer Peer-reviewed

    Mohammad Razuanul Hogue, Toru Ishizuka, Keiichi Inoue, Rei Abe-Yoshizumi, Hiroyuki Igarashi, Takaaki Mishima, Hideki Kandori, Hiromu Yawo

    PLOS ONE　11　(11)　e0166820　2016/11

    DOI： 10.1371/journal.pone.0166820 　

    ISSN： 1932-6203

  16. A Novel Reporter Rat Strain That Conditionally Expresses the Bright Red Fluorescent Protein tdTomato Peer-reviewed

    Hiroyuki Igarashi, Kyo Koizumi, Ryosuke Kaneko, Keiko Ikeda, Ryo Egawa, Yuchio Yanagawa, Shin-ichi Muramatsu, Hiroshi Onimaru, Toru Ishizuka, Hiromu Yawo

    PLOS ONE　11　(5)　e0155687　2016/05

    DOI： 10.1371/journal.pone.0155687 　

    ISSN： 1932-6203

  17. A Phox2b BAC Transgenic Rat Line Useful for Understanding Respiratory Rhythm Generator Neural Circuitry Peer-reviewed

    Keiko Ikeda, Masanori Takahashi, Shigeru Sato, Hiroyuki Igarashi, Toru Ishizuka, Hiromu Yawo, Satoru Arata, E. Michelle Southard-Smith, Kiyoshi Kawakami, Hiroshi Onimaru

    PLOS ONE　10　(7)　e0132475　2015/07

    DOI： 10.1371/journal.pone.0132475 　

    ISSN： 1932-6203

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Misc. 4

  1. A brain-wide exploration of sex differences in neural circuits carrying interoceptive information.

    M. Goto, M. Yamakawa, K. Ohbayashi, Y. Iwasaki, T. Sasaki, H. Igarashi

    2025/09

    DOI： 10.1016/j.jphyss.2025.100040 　

  2. Activation of Phox2B-positive neurons in the dorsal medulla induced sucking and hiccup

    Iizuka Makito, Ikeda Keiko, Igarashi Hiroyuki, Kobayashi Kazuto, Onimaru Hiroshi, Izumizaki Masahiko

    The Journal of Physiological Sciences　73　(Suppl.1)　113-113　2023/05

    Publisher: (一社)日本生理学会

    ISSN： 1880-6546

    eISSN： 1880-6562

  3. 孤束核のPhox2b陽性ニューロンは吸啜リズムの発現に必須である(Phox2b-positive neurons located in the solitary nucleus is essential to trigger sucking)

    Iizuka Makito, Ikeda Keiko, Igarashi Hiroyuki, Kobayashi Kazuto, Onimaru Hiroshi, Izumizaki Masahiko

    The Journal of Physiological Sciences　71　(Suppl.1)　165-165　2021/08

    Publisher: (一社)日本生理学会

    ISSN： 1880-6546

    eISSN： 1880-6562

  4. Generation of transgenic mice to analyze the role of Nurr1 and Nur77 genes in the adult brain

    Takayuki Hatanaka, Yasuhito Hatanaka, Takehito Ito, Hiroyuki Igarashi, Hirofumi Tokuoka, Shin-ichi Muramatsu, Tsuyoshi Endo, Ken Hirosaki, Akihiko Takasaki, Nobuhiro Hayashi, Daniel Metzger, Hiroshi Ichinose

    NEUROSCIENCE RESEARCH　71　E213-E214　2011

    DOI： 10.1016/j.neures.2011.07.928 　

    ISSN： 0168-0102

Books and Other Publications 1

  1. ラットを用いたオプトジェネティクス（光遺伝学）

    八尾 寛、深澤有吾、冨田浩史、五十嵐敬幸

    日本実験動物協会情報誌 LABIO 21 No.80　2020/05

Industrial Property Rights 1

  1. シャープペンシル

    五十嵐敬幸, 阿部洋一, 荒川康平

    Property Type: Patent

Research Projects 6

  1. 全身信号計測法によるストレス適応メカニズムの解明

    五十嵐 敬幸

    Offer Organization: 日本学術振興会

    System: 科学研究費助成事業

    Category: 若手研究

    Institution: 東北大学

    2024/04 - 2027/03

  2. 左右非対称性と性差から捉える中枢ストレス応答系の制御機構

    五十嵐敬幸

    Offer Organization: 公益財団法人鈴木謙三記念医科学応用研究財団

    System: 令和7年度調査研究助成金

    2026/01 - 2026/12

  3. 高周波数超音波を用いた４次元脳血管イメージングによる、マウス脳梗塞モデルの血行動態評価

    研究代表者 石井琢郎(東北大・医工学研究科), 研究協力者 熊井萌(東北大・医学研究科)、金子悦久(東北大・医工学研究科)、安藤大祐(東北大・医学研究科)、五十嵐敬幸(東北大・薬学研究科)、新妻邦泰(東北大・医学研究科)、遠藤英徳(東北大・医学研究科)、西條芳文(東北大・医工学研究科)

    Offer Organization: 日本医療研究開発機構

    System: 「橋渡し研究プログラム」研究開発課題 異分野融合型研究開発推進支援事業

    2025/09 - 2026/03

  4. 齧歯動物モデルを使った全身流体情報と生理反応の網羅的記録法の開発

    石井琢郎、五十嵐敬幸

    Offer Organization: 東北大学

    System: 学際科学フロンティア研究所 学際研究共創プログラム

    2024/05 - 2026/03

  5. 興奮性-抑制性バランスの変化が生み出す個体のストレス適応メカニズムの解明

    五十嵐敬幸、瀧川健司

    Offer Organization: 東北大学

    System: はばたく若手支援事業

    2024/09 - 2025/08

  6. 室傍核による内受容感覚の統合と全身性ストレス応答メカニズムの解明

    五十嵐 敬幸、増田 雄太

    Offer Organization: 国立研究開発法人科学技術振興機構（JST）

    System: CREST若手チャレンジ

    2024/04 - 2025/03

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Teaching Experience 6

  1. Functional Morphology II　東北大学

  2. 学問論演習　東北大学

  3. Pharmacology 3　Tohoku University

  4. 医療薬学実習　東北大学薬学部

  5. Introductory Science Experiments　Tohoku University

  6. Physiology 4730B – DNA cloning　University of Western Ontario

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Academic Activities 6

  1. 第64回日本薬学会 東北支部大会（生物系・座長）

    2025/10/18 - 2025/10/18

    Activity type: Academic society, research group, etc.

  2. 学術変革領域A「適応回路センサス」共催：脳の適応機能に挑む若手研究者のポスター発表の集い

    2025/07/24 - 2025/07/24

    Activity type: Academic society, research group, etc.

  3. 東北大学薬学研究科 第600回薬学セミナー

    2025/04/22 - 2025/04/22

    Activity type: Other

  4. NEURO2024 若手育成道場 若手座長

    2024/07/24 - 2024/07/24

    Activity type: Academic society, research group, etc.

  5. Physiology and Pharmacology Research Day, Poster Judge

    2020/10/03 - 2020/10/03

    Activity type: Academic society, research group, etc.

  6. 第9回光操作研究会 セッション4 副座長

    2017/10/21 - 2017/10/22

    Activity type: Academic society, research group, etc.

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Other 2

  1. 論文査読（Neuroscience research）

  2. 論文査読（Journal of pharmacological sciences）